Personalized blood flow constraint rehab training (PBFR) is a game-changing injury healing therapy that is producing drastically positive outcomes: Lessen atrophy and loss of strength from disuse and non-weight bearing after injuries Increase strength with only 30% loads Boost hypertrophy with only 30% loads Enhance muscle endurance in 1/3 the time Enhance muscle protein synthesis in the senior Improve strength and hypertrophy after surgery Enhance muscle activation Boost development hormonal agent responses.

Muscle weakness commonly happens in a range of conditions and pathologies. High load resistance training has been revealed to be the most effective methods in improving muscular strength and acquiring muscle hypertrophy. The problem that exists is that in specific populations that require muscle enhancing eg Chronic Discomfort Clients or post-operative clients, high load and high intensity workouts may not be clinically appropriate.

It has been used in the health club setting for some time but it is getting appeal in scientific settings. BFR training was initially developed in the 1960's in Japan and understood as KAATSU training.

It can be applied to either the upper or lower limb. The cuff is then pumped up to a specific pressure with the aim of acquiring partial arterial and total venous occlusion. Muscle hypertrophy is the increase in size of the muscle as well as a boost of the protein material within the fibers.

Muscle tension and metabolic tension are the two main factors accountable for muscle hypertrophy. Mechanical Tension & Metabolic Tension [edit edit source] When a muscle is placed under mechanical stress, the concentration of anabolic hormone levels increase. The activation of myogenic stem cells and the raised anabolic hormonal agents result in protein metabolism and as such muscle hypertrophy can take place.

Insulin-like development element and development hormonal agent are responsible for increased collagen synthesis after workout and help muscle recovery. Development hormonal agent itself does not directly trigger muscle hypertrophy however it aids muscle recovery and thereby potentially assists in the muscle reinforcing process. The accumulation of lactate and hydrogen ions (eg in hypoxic training) more increases the release of growth hormonal agent.

Myostatin controls and prevents cell development in muscle tissue. Resistance training results in the compression of blood vessels within the muscles being trained.

This results in an increase in anaerobic lactic metabolism and the production of lactate. When there is blood pooling and an accumulation of metabolites cell swelling happens. This swelling within the cells triggers an anabolic reaction and results in muscle hypertrophy. The cell swelling may actually cause mechanical tension which will then activate the myogenic stem cells as gone over above.

The cuff is put proximally to the muscle being exercise and low strength workouts can then be carried out. Due to the fact that the outflow of blood is restricted using the cuff capillary blood that has a low oxygen material collects and there is an increase in protons and lactic acid. The exact same physiological adjustments to the muscle (eg release of hormones, hypoxia and cell swelling) will happen throughout the BFR training and low intensity exercise as would accompany high strength workout.

( 1) Low intensity BFR (LI-BFR) leads to an increase in the water content of the muscle cells (cell swelling). It also speeds up the recruitment of fast-twitch muscle fibers. It is likewise hypothesized that when the cuff is removed a hyperemia (excess of blood in the blood vessels) will form and this will trigger more cell swelling.

These increases were similar to gains acquired as an outcome of high-intensity workout without BFR A research study comparing (1) high intensity, (2) low strength, (3) high and low strength with BFR and (4) low intensity with BFR. While all 4 exercise regimes produced increases in torque, muscle activations and muscle endurance over a 6 week period - the high strength (group 1) and BFR (groups 3 and 4) produced the best effect size and were comparable to each other.